Calcific tendinitis of the rotator cuff (CTR) is common in women aged 30–60 years, accounting for 5–40% of the causes of shoulder pain. The supraspinatus tendon accounts for about 80%, the infraspinatus tendon accounts for 15%, and the subscapularis tendon accounts for 5% [1]. About 20% of patients may have no clinical symptoms, and about 50% of patients have no obvious cause. The etiology is still unclear and may be related to degeneration, trauma, hypoxia, endocrine, metabolic disorders, etc. [2, 3]. Due to the deposition of hydroxyapatite [Ca10(PO4)6(OH)2] and inflammatory stimulation, the onset is sudden, knife-like severe pain and limited shoulder joint activity [4]. There are many theories about the pathological changes of CTR, and the theory of reactive calcification proposed by Uhthoff et al. [4] has been widely recognized.