Larva migrans is a zoonotic helminthosis in humans caused by the abnormal migration of animal nematode larvae within visceral or cutaneous tissues that typically occurs after the inadvertent ingestion of infective eggs or larvae through contaminated food and water. As larvae undergo somatic migration through various internal tissues and organs, they trigger host inflammatory responses, often resulting in a range of clinical symptoms commonly called as larva migrans syndrome. The primary causative agents for visceral larva migrans include Toxocara spp., Angiostrongylus spp., Gnathostoma spinigerum, and Baylisascaris procyonis, while the cutaneous form of larva migrans results from the percutaneous migration of animal hookworm larvae. Children are particularly vulnerable due to their tendency for geophagia and poor understanding of hygiene. While many cases remain asymptomatic, heavy parasitic loads or infections affecting vital organs can result in substantial morbidity. The diagnosis of larval migraine is primarily based on serology-based testing, with elevated immunoglobulin E (IgE) levels serving as adjunct markers that may indicate disease severity and outcomes. Therapeutic management typically includes the administration of anthelmintics, alongside corticosteroids, for symptomatic relief of itching and inflammation. However, effective prevention strategies must emphasise reducing exposure to infection sources, implementing routine deworming protocols for pets, and fostering public health education initiatives.