NSTEMI, a variant of acute coronary syndrome (ACS), presents with myocardial damage evidenced by elevated troponins, yet lacks the hallmark ST-segment elevation on ECG. The pathophysiology of NSTEMI involves a mismatch between myocardial oxygen supply and demand. This imbalance is often due to partial occlusion of a coronary artery by a thrombus superimposed on a disrupted atherosclerotic plaque. However, other mechanisms such as plaque instability, vasospasm, myocarditis, or even non-atherosclerotic causes like pulmonary embolism can also precipitate NSTEMI. Understanding these varied etiologies is crucial for distinguishing NSTEMI from other forms of ACS and tailoring appropriate management strategies [1–3] [Ref: Algorithm 64.1].