Post-traumatic lymphedema (PTL) is a complex and frequently underrecognized sequela resulting from substantial trauma to the limbs or trunk. It is defined by the persistent accumulation of protein-rich interstitial fluid, arising from disruption or damage to the lymphatic pathways secondary to injury. This pathologic fluid retention contributes to progressive limb swelling, cutaneous changes, and varying degrees of functional impairment. Distinct from primary lymphedema, which is linked to congenital or developmental lymphatic anomalies, and the more widely acknowledged secondary lymphedema related to oncologic interventions such as lymphadenectomy and radiotherapy, PTL stems directly from mechanical trauma. This may include high-energy injuries such as crush trauma, fractures, major soft tissue disruptions, thermal burns, or inadvertent lymphatic injury during reconstructive or limb-salvaging surgical procedures. The lymphatic vessels, due to their fragile and superficial nature, are highly susceptible to direct injury, external compression from hematomas, or obliteration due to fibrotic remodeling and inflammatory responses triggered by the initial insult [1].