Motor epilepsies present a significant clinical challenge, particularly in cases where pharmacological treatments have failed. Seizures originating primarily from the motor cortex often manifest as tonic or clonic movements affecting the contralateral hemibody, which can lead to falls, accidents, and significant motor impairment. In a clinical setting, frequent motor seizures not only restrict voluntary motor activity but also contribute to loss of independence and necessitate continuous supervision and assistance from caregivers, ultimately impacting the patient’s and caregiver’s quality of life. Given the potential for severe and irreversible motor deficits associated with surgical resection of an epileptogenic zone (EZ) in the motor cortex, deep brain stimulation (DBS) of motor-related basal ganglia structures has emerged as a potential alternative therapeutic strategy for drug-resistant motor epilepsy.