Spinal cord injury (SCI) causes a variety of sequelae, including movement loss, decrease in muscle mass of affected limbs, and urinary tract disturbances. Damage to the injured neural tissue results in acute neuronal loss followed by a long-lasting cascade of secondary cytotoxic events. Although it is well established that sequential molecular events can lead to cellular apoptosis of surviving neurons, little is known about the relation between different early phases of neural damage and functional factors. Thus, this study aimed to characterize spinal shock and the acute phase of traumatic SCI due to thoracic compression in female rats, evaluating neural and functional factors from the 1st to 14th postoperative day (POD). Five female Wistar rats underwent the SCI procedure using a compression model, and were evaluated to locomotion using the BBB (Basso, Beatie and Bresnahan) locomotion classification scale, collection of residual urine volume, followed by analysis of muscle trophism. Immunofluorescence images of the spinal cord for neuronal suvive and activity were quantified through simple morphometric counting. Statistical analysis showed increased BBB score in 14 days post-injury compared to the 1 and 7 POD. The residual urine volume increased in 7 POD followed by significant reduction in 14 days, while analysis of the soleus muscle revealed a decrease of the weight in 7 and 14 POD compared to the 1 day. Morphological analysis of the dorsal root showed a little representative change in the first days after SCI. Therefore, the results suggest that functional changes in the acute phase of the clip-compression model are not related to morphology in the site injury.