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Background <p>Dystrophic calcification affecting cardiac structures secondary to hyperadrenocorticism (HAC) is rare in dogs. This case report describes the clinical, imaging, surgical, and pathological findings of a 9-year-old spayed female Maltese dog with severe mitral regurgitation and HAC-related systemic calcification presenting as an intracardiac calcified mass.</p> Case presentation <p>Severe mitral valve thickening and a mobile hyperechoic mass attached to the chordae tendineae were detected on echocardiography. Mitral valvuloplasty was performed, and the calcified mass was excised. Histopathology confirmed a non-neoplastic calcified lesion. Computed tomography revealed extensive calcification in various tissues. The dog developed postoperative aspiration pneumonia and died on postoperative day 2.</p> Conclusions <p>This case highlights the potential for intracardiac dystrophic calcification in dogs with HAC and severe mitral valve disease.</p>

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Calcified mass attached to mitral valve chordae tendineae in a dog with hyperadrenocorticism: A case report

  • 期刊论文

  • 出版模式：

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  • 发表日期：

    2025年12月5日

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  • Tomoki Wada,
  • Ryosuke Takemura,
  • Hideki Yotsuida,
  • Tomoya Okano,
  • Takuya Mori

摘要

Background

Dystrophic calcification affecting cardiac structures secondary to hyperadrenocorticism (HAC) is rare in dogs. This case report describes the clinical, imaging, surgical, and pathological findings of a 9-year-old spayed female Maltese dog with severe mitral regurgitation and HAC-related systemic calcification presenting as an intracardiac calcified mass.

Case presentation

Severe mitral valve thickening and a mobile hyperechoic mass attached to the chordae tendineae were detected on echocardiography. Mitral valvuloplasty was performed, and the calcified mass was excised. Histopathology confirmed a non-neoplastic calcified lesion. Computed tomography revealed extensive calcification in various tissues. The dog developed postoperative aspiration pneumonia and died on postoperative day 2.

Conclusions

This case highlights the potential for intracardiac dystrophic calcification in dogs with HAC and severe mitral valve disease.