Scholarlink

Abstract <p>The study was aimed at exploring the prevalence and characteristics of the postactivation effect (PAE) in deltoid muscles (DM) in individuals with Parkinson’s disease (PD) and its possible correlation with PD symptoms using surface electromyography (sEMG), in the off- and on-medication phases. Ten of 29 of individuals with PD (34.5%) were PAE-positive as they triggered PAE. PAE duration was different on the right and left sides in 5 of 8 PDs (mean duration 41–47 s), and the latency was 1.7 s The PAE prevalence, amplitude and mean frequency of sEMG during PAE did not differ between the medication phases. The UPDRS-based scores of bradykinesia were lower in the PAE-positive than in PAE-negative individuals (<i>p</i> &lt; 0.01). In addition, bradykinesia scores were inversely correlated with PAE duration, and akinesia scores were inversely correlated with sEMG amplitude during PAE. In conclusion, PAE triggering in PPD, its amplitude, and duration are likely inhibited by mechanisms associated with bradykinesia and akinesia.</p>

错误：搜索内容不能为空，请输入英文关键词
错误：关键词超出字数限制，请精简
高级检索

Low Prevalence of Postactivation Effect (Kohnstamm Phenomenon) in People with Parkinson’s Disease is Associated with Bradykinesia

  • 期刊论文

  • 发表日期：

    2025年12月1日

查看全文
  • A. Meigal,
  • L. Gerasimova-Meigal,
  • E. Antonen,
  • A. Peskova

摘要

Abstract

The study was aimed at exploring the prevalence and characteristics of the postactivation effect (PAE) in deltoid muscles (DM) in individuals with Parkinson’s disease (PD) and its possible correlation with PD symptoms using surface electromyography (sEMG), in the off- and on-medication phases. Ten of 29 of individuals with PD (34.5%) were PAE-positive as they triggered PAE. PAE duration was different on the right and left sides in 5 of 8 PDs (mean duration 41–47 s), and the latency was 1.7 s The PAE prevalence, amplitude and mean frequency of sEMG during PAE did not differ between the medication phases. The UPDRS-based scores of bradykinesia were lower in the PAE-positive than in PAE-negative individuals (p < 0.01). In addition, bradykinesia scores were inversely correlated with PAE duration, and akinesia scores were inversely correlated with sEMG amplitude during PAE. In conclusion, PAE triggering in PPD, its amplitude, and duration are likely inhibited by mechanisms associated with bradykinesia and akinesia.