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<p>A 56-year-old man with cognitive decline and right hemiparesis for 1 week.&#xa0;Neuroimaging revealed a bilateral high intensity signal in the corticomedullary junction&#xa0;and left temporal lobe on diffusion-weighted imaging (DWI) (figure A and C), a&#xa0;corresponding low signal on ADC (figure B and D), a slightly hyperperfusion in left&#xa0;temporal lobe (figure E) and high-intensity signal in bilateral middle cerebellar&#xa0;peduncles on fluid-attenuated inversion recovery (figure G). Postcontrast magnetic&#xa0;resonance imaging showed enhancement of the left occipito-parietal cortex (figure H).&#xa0;A diagnosis of NIID was established based on skin biopsy and genetic testing.&#xa0;Temporal lobe hyperperfusion, occipito-parietal cortical enhancement and a high signal&#xa0;in bilateral middle cerebellar peduncles may be a diagnostic clue for NIID in patients&#xa0;mimic stroke&#xa0;[1].</p>

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Teaching NeuroImage: an imaging clue for neuronal intranuclear inclusion disease

  • 期刊论文

  • 发表日期：

    2026年3月23日

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  • Jianhua Yang,
  • Qing Liu,
  • Xianghong Liu

摘要

A 56-year-old man with cognitive decline and right hemiparesis for 1 week. Neuroimaging revealed a bilateral high intensity signal in the corticomedullary junction and left temporal lobe on diffusion-weighted imaging (DWI) (figure A and C), a corresponding low signal on ADC (figure B and D), a slightly hyperperfusion in left temporal lobe (figure E) and high-intensity signal in bilateral middle cerebellar peduncles on fluid-attenuated inversion recovery (figure G). Postcontrast magnetic resonance imaging showed enhancement of the left occipito-parietal cortex (figure H). A diagnosis of NIID was established based on skin biopsy and genetic testing. Temporal lobe hyperperfusion, occipito-parietal cortical enhancement and a high signal in bilateral middle cerebellar peduncles may be a diagnostic clue for NIID in patients mimic stroke [1].